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Serum GPNMB levels increased with the progression of NAFLD activity score and liver fibrosis stage
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Overall, lipid peroxidation emerges as a central driver in the pathogenesis and progression of pulmonary fibrosis: it not only disrupts AT2 cell barrier function and promotes aberrant fibroblast activation thereby sustaining a persistent oxidative injury environment -but also likely interacts with ferroptosis and metabolic reprogramming to collectively fuel a self-amplifying fibrotic cycle
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